Translations:Dave Johnson's Proletariat Protocol/25/en

It appears that at least in the case of SOD1 fALS, the attack is going to proceed because of a fundamental defect in the astroglia, without dependence on release of proinflammatory cytokines by damaged neurons to maintain the attack. Therefore a protocol such as the Prole Prote, although it may prolong life somewhat in an SOD1 sALS patient, is not going to halt the disease much less make recovery possible. This is more than just a limitation of the Prole Prote, it indicates a fundamental inability of the G93a mouse model to be used for testing therapeutics intended for a sporadic ALS patient population.