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"We treated each group of 11 ALS model mice with EGCG ... and one group of 11 ... (control group) intraorally every day after 60 days of age (presymptomatic treatment). The treatment of more than 2.9 microg EGCG/g body weight significantly prolonged the symptom onset and life span, preserved more survival signals, and attenuated death signals. These data suggest that EGCG could be a potential therapeutic candidate for ALS as a disease-modifying agent."{{#pmid: 16356650 |Koh2006}} | "We treated each group of 11 ALS model mice with EGCG ... and one group of 11 ... (control group) intraorally every day after 60 days of age (presymptomatic treatment). The treatment of more than 2.9 microg EGCG/g body weight significantly prolonged the symptom onset and life span, preserved more survival signals, and attenuated death signals. These data suggest that EGCG could be a potential therapeutic candidate for ALS as a disease-modifying agent."{{#pmid: 16356650 |Koh2006}} | ||
− | '' | + | ''n conclusion, EGCG leads to a decrease in oxidative stress levels, leading to motor neuron protection in the organotypic culture of a rat spinal cord; however, EGCG does not alter iron metabolism protein expression regulation.'' {{#pmid:28677731|che2017}} |
==References== | ==References== | ||
[[Category:Supplement data pages]] | [[Category:Supplement data pages]] |